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  1. 学部学科区分一覧
  2. 医学部・医学系研究科・医学部附属病院
  1. 資料タイプ一覧
  2. 学術雑誌論文
  1. 鳥取大学の刊行物
  2. Yonago Acta Medica
  3. 52
  4. 4

Recent Advances in Ultrasound Imaging of Breast Lesions

https://repository.lib.tottori-u.ac.jp/records/5032
https://repository.lib.tottori-u.ac.jp/records/5032
1810c5a5-4b59-4de6-8c81-57cd3647a313
名前 / ファイル ライセンス アクション
52_115-120(1).pdf 52_115-120(1).pdf (290.5 kB)
Item type 学術雑誌論文 / Journal Article(1)
公開日 2018-06-22
タイトル
タイトル Recent Advances in Ultrasound Imaging of Breast Lesions
言語 en
言語
言語 eng
キーワード
主題 breast
キーワード
主題 cancer
キーワード
主題 elastography
キーワード
主題 mass screening
キーワード
主題 ultrasonography
キーワード
言語 en
主題 breast
キーワード
言語 en
主題 cancer
キーワード
言語 en
主題 elastography
キーワード
言語 en
主題 mass screening
キーワード
言語 en
主題 ultrasonography
資源タイプ
資源タイプ journal article
著者 Hirooka, Yasuaki

× Hirooka, Yasuaki

WEKO 752
e-Rad 40243399
研究者総覧鳥取大学 100000228

Hirooka, Yasuaki

en Hirooka, Yasuaki

Search repository
Aika, Noriko

× Aika, Noriko

WEKO 18472

Aika, Noriko

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Ishisugi, Takuya

× Ishisugi, Takuya

WEKO 18473

Ishisugi, Takuya

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Ohguri, Masayoshi

× Ohguri, Masayoshi

WEKO 2761
研究者総覧鳥取大学 100001721

Ohguri, Masayoshi

ja-Kana オグリ, マサヨシ

en Ohguri, Masayoshi

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Nagashima, Chiharu

× Nagashima, Chiharu

WEKO 18474

Nagashima, Chiharu

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Morishita, Shota

× Morishita, Shota

WEKO 18475

Morishita, Shota

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Kato, Yosuke

× Kato, Yosuke

WEKO 18476

Kato, Yosuke

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Fukuda, Chisako

× Fukuda, Chisako

WEKO 389
e-Rad 60116366

Fukuda, Chisako

en Fukuda, Chisako

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Aika, Noriko

× Aika, Noriko

WEKO 18477

en Aika, Noriko

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Ishisugi, Takuya

× Ishisugi, Takuya

WEKO 18478

en Ishisugi, Takuya

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Nagashima, Chiharu

× Nagashima, Chiharu

WEKO 18479

en Nagashima, Chiharu

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Morishita, Shota

× Morishita, Shota

WEKO 18480

en Morishita, Shota

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Kato, Yosuke

× Kato, Yosuke

WEKO 18481

en Kato, Yosuke

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著者所属
値 Department of Pathobiological Science and Technology, School of Health Science, Tottori University Faculty of Medicine
著者所属
値 Department of Pathobiological Science and Technology, School of Health Science, Tottori University Faculty of Medicine
著者所属
値 Department of Pathobiological Science and Technology, School of Health Science, Tottori University Faculty of Medicine
著者所属
値 Department of Pathobiological Science and Technology, School of Health Science, Tottori University Faculty of Medicine
著者所属
値 Department of Pathobiological Science and Technology, School of Health Science, Tottori University Faculty of Medicine
著者所属
値 Department of Pathobiological Science and Technology, School of Health Science, Tottori University Faculty of Medicine
著者所属
値 Department of Pathobiological Science and Technology, School of Health Science, Tottori University Faculty of Medicine
著者所属
値 Department of Pathobiological Science and Technology, School of Health Science, Tottori University Faculty of Medicine
抄録
内容記述タイプ Other
内容記述 Helminth parasites are known to elicit the immune response towards T helper 2 (Th2)-type, characterized by Th2 related cytokines, that typically include interleukin-4 (IL-4), IL-5 and IL-13. In this review we will describe the mechanisms involved in helminth induced Th2 immune response. Intestinal epithelial cells (IECs) produce thymic stromal lymphopoietin (TSLP), which is both necessary and sufficient for the initiation of Th2 cytokine-driven inflammation. IL-33 mRNA is expressed early during parasite infection and IL-33 binds ST2 receptor, both of which are associated with optimal CD4+ Th2 polarization. Following innate immune cell recognition, basophils and mast cell can secrete Th2 type cytokines that are thought to contribute to CD4+ Th2 differentiation. Additionaly, dendritic cell conditioned with some helminth products can promote CD4+ Th2 differentiation. Alternatively activated macrophages, activated by the Th2 cytokines IL-4 and IL-13 in parasitic infections, contribute to the host protective response: control of Th1-type inflammation, wound healing and worm expulsion. Experimentally, helminths have been associated with protection against a number of autoimmune disorders, including inflammatory bowel diseases and type 1 diabetes. It may be a novel strategy to ameliorate autoimmune inflammation by expanding and activating the Th2 response originated from parasites.
書誌情報 Yonago Acta medica
en : Yonago Acta medica

巻 52, 号 4, p. 115-120, 発行日 2009-12
出版者
出版者 Tottori University Faculty of Medicine
ISSN
収録物識別子タイプ ISSN
収録物識別子 13468049
書誌レコードID
収録物識別子タイプ NCID
収録物識別子 AA00892882
権利
権利情報 Yonago Acta medica 編集委員会
情報源
関連名称 Yonago Acta medica. 2009, 52(4), 115-120
著者版フラグ
出版タイプ VoR
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