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  1. 学部学科区分一覧
  2. 医学部・医学系研究科・医学部附属病院
  1. 資料タイプ一覧
  2. 学術雑誌論文
  1. 鳥取大学の刊行物
  2. Yonago Acta Medica
  3. 50
  4. 3

Cardiac Preconditioning by Anesthetic Agents: Roles of Volatile Anesthetics and Opioids in Cardioprotection

https://repository.lib.tottori-u.ac.jp/records/5057
https://repository.lib.tottori-u.ac.jp/records/5057
cd76a298-b295-4bc6-a931-6b424785b668
名前 / ファイル ライセンス アクション
50_045-055.pdf 50_045-055.pdf (432.6 kB)
Item type 学術雑誌論文 / Journal Article(1)
公開日 2018-06-22
タイトル
言語 en
タイトル Cardiac Preconditioning by Anesthetic Agents: Roles of Volatile Anesthetics and Opioids in Cardioprotection
言語
言語 eng
キーワード
主題 ATP-sensitive potassium channel
キーワード
主題 ischemic preconditioning
キーワード
主題 pharmacological preconditioning
キーワード
主題 volatile anesthetic
キーワード
主題 opioid
キーワード
言語 en
主題 ATP-sensitive potassium channel
キーワード
言語 en
主題 ischemic preconditioning
キーワード
言語 en
主題 pharmacological preconditioning
キーワード
言語 en
主題 volatile anesthetic
キーワード
言語 en
主題 opioid
資源タイプ
資源タイプ journal article
著者 Inagaki, Yoshimi

× Inagaki, Yoshimi

WEKO 938
e-Rad 40184717
研究者総覧鳥取大学 100000092

Inagaki, Yoshimi

en Inagaki, Yoshimi

Search repository
著者所属
Division of Anesthesiology and Critical Care Medicine, Department of Surgery, School of Medicine, Tottori University Faculty of Medicine
抄録
内容記述タイプ Other
内容記述 Cardiac preconditioning is the most potent and consistently reproducible method of protecting heart tissue against myocardial ischemia-reperfusion injury. This review discussed about the signaling and amplification cascades from either ischemic preconditioning stimulus or pharmacological preconditioning stimulus, the putative end-effectors and the mechanisms involved in cellular protection. The pharmacological preconditioning induced by volatile anesthetics and opioids is very similar to the ischemic preconditioning. It includes activation of G-protein-coupled receptors, multiple protein kinases and ATP-sensitive potassium channels (KATP channels). Volatile anesthetics prime the activation of the sarcolemmal and mitochondrial KATP channels, which are the putative end-effectors of preconditioning, by stimulation of adenosine receptors and subsequent activation of protein kinase C (PKC) and by increased formation of nitric oxide and free oxygen radicals. Similarly, opioids activate δ- and κ-opioid receptors leading to activation of PKC. The open state of the mitochondrial KATP channel and sarcolemmal KATP channel ultimately induces cytoprotection by decreasing Ca2+ overload in the cytosol and mitochondria.
書誌情報 Yonago Acta medica
en : Yonago Acta medica

巻 50, 号 3, p. 45-55, 発行日 2007-09
出版者
出版者 Tottori University Faculty of Medicine
ISSN
収録物識別子タイプ ISSN
収録物識別子 13468049
書誌レコードID
収録物識別子タイプ NCID
収録物識別子 AA00892882
権利
権利情報 Yonago Acta medica 編集委員会
情報源
関連名称 Yonago Acta medica. 2007, 50(3), 45-55
著者版フラグ
出版タイプ VoR
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